• Complex
  • Title
  • Author
  • Keyword
  • Abstract
  • Scholars
High Impact Results & Cited Count Trend for Year Keyword Cloud and Partner Relationship

Query:

学者姓名:闫剑群

Refining:

Source

Submit Unfold

Language

Submit

Clean All

Export Sort by:
Default
  • Default
  • Title
  • Year
  • WOS Cited Count
  • Impact factor
  • Ascending
  • Descending
< Page ,Total 11 >
Angiotensin II facilitates GABAergic neurotransmission at postsynaptic sites in rat amygdala neurons SCIE PubMed Scopus
期刊论文 | 2018 , 133 , 334-344 | NEUROPHARMACOLOGY
Abstract&Keyword Cite

Abstract :

The central nucleus of the amygdala (CeA) is critical in the regulation of sodium appetite. Angiotensin II (Ang II) is important in the generation of sodium appetite and may function as a neurotransmitter or modulator to affect the synaptic transmission and the excitability of neurons. However, the role of Ang II in the CeA remains unclear. In this study, we determined the effects of Ang II on the excitatory and inhibitory synaptic inputs to the CeA neurons in brain slices with whole-cell patch-clamp recordings. Ang II (0.5-5 mu M) significantly potentiated the amplitude of spontaneous GABAergic inhibitory postsynaptic currents (IPSCs) in a concentration-dependent manner. Ang II (2 mu M) significantly increased the amplitude of miniature GABAergic inhibitory postsynaptic currents (mIPSCs) without affecting the frequency. This effect was blocked by Ang II type 1 (AT(1)) receptor antagonist, losartan. One mM guanosine 5'-O-(-2-thiodiphosphate) (GDP-beta-s) in the pipette internal solution eliminated the facilitatory effect of Ang II on GABAergic synaptic transmission. In contrast, Ang II had no effect on the spontaneous glutamatergic excitatory postsynaptic currents (EPSCs) and did not alter the frequency and amplitude of miniature EPSCs at concentrations that facilitated IPSCs. Furthermore, Ang II decreased the firing activity of CeA neurons, and this effect was abolished by losartan and GDP-beta-s. In addition, Ang II failed to inhibit CeA neurons in the presence of bicuculline. These data provide substantial new evidence that Ang II inhibits the CeA neurons by facilitation of GABAergic synaptic input efficacy through activation of postsynaptic ATE receptors. (C) 2018 Elsevier Ltd. All rights reserved.

Cite:

Copy from the list or Export to your reference management。

GB/T 7714 Hu, Bo , Qiao, Hu , Cao, Tian et al. Angiotensin II facilitates GABAergic neurotransmission at postsynaptic sites in rat amygdala neurons [J]. | NEUROPHARMACOLOGY , 2018 , 133 : 334-344 .
MLA Hu, Bo et al. "Angiotensin II facilitates GABAergic neurotransmission at postsynaptic sites in rat amygdala neurons" . | NEUROPHARMACOLOGY 133 (2018) : 334-344 .
APA Hu, Bo , Qiao, Hu , Cao, Tian , Sun, Bo , Luo, Xiao , Jia, Ru et al. Angiotensin II facilitates GABAergic neurotransmission at postsynaptic sites in rat amygdala neurons . | NEUROPHARMACOLOGY , 2018 , 133 , 334-344 .
Export to NoteExpress RIS BibTex
Prenatal high-fat diet alters placental morphology, nutrient transporter expression, and mtorc1 signaling in rat SCIE PubMed Scopus
期刊论文 | 2017 , 25 (5) , 909-919 | OBESITY | IF: 4.042
WoS CC Cited Count: 1
Abstract&Keyword Cite

Abstract :

ObjectiveThis study aimed to determine how the rat placenta and fetus respond to maternal high-fat (HF) diet during gestation and to identify the possible mechanisms. MethodsPregnant Sprague-Dawley rats were fed with standard chow (13.5% fat) or HF (60% fat) diet during gestation. Placentas were collected on gestation day 21. ResultsHF dams had greater fat mass, higher plasma leptin, lower plasma adiponectin, and impaired glucose tolerance during pregnancy. The placental labyrinth thickness was reduced in both male and female fetuses of HF dams. In HF male placentas, glucose transporter 3 gene expression, system A amino acid transporter (SNAT) 2 gene expression, and SNAT2 protein expression were increased through the activation of the mTORC1 4EBP1 branch. In HF female placentas, gene expression of insulin-like growth factor 2 (IGF2) and IGF2 receptor was elevated compared to placentas of females fed standard chow. Although male and female placentas responded differently to prenatal HF diet exposure, both male and female fetal weight was not altered by maternal HF diet. ConclusionsPlacenta responds and adapts to maternal metabolic changes by altering placental layer thickness, mTORC1 signaling, expression of nutrient transporters, and growth factors in a sex-specific manner.

Cite:

Copy from the list or Export to your reference management。

GB/T 7714 Song, Lin , Sun, Bo , Boersma, Gretha J. et al. Prenatal high-fat diet alters placental morphology, nutrient transporter expression, and mtorc1 signaling in rat [J]. | OBESITY , 2017 , 25 (5) : 909-919 .
MLA Song, Lin et al. "Prenatal high-fat diet alters placental morphology, nutrient transporter expression, and mtorc1 signaling in rat" . | OBESITY 25 . 5 (2017) : 909-919 .
APA Song, Lin , Sun, Bo , Boersma, Gretha J. , Cordner, Zachary A. , Yan, Jianqun , Moran, Timothy H. et al. Prenatal high-fat diet alters placental morphology, nutrient transporter expression, and mtorc1 signaling in rat . | OBESITY , 2017 , 25 (5) , 909-919 .
Export to NoteExpress RIS BibTex
Taste sensitivity to sucrose is lower in outbred Sprague-Dawley phenotypic obesity-prone rats than obesity-resistant rats SCIE PubMed Scopus
期刊论文 | 2017 , 489 (2) , 155-163 | BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS | IF: 2.559
Abstract&Keyword Cite

Abstract :

The purpose of the present study was to better understand the role of sweet taste perception in dietary behavior and body weight in outbred Sprague-Dawley phenotypic obesity-prone and obesity-resistant rats by measuring sucrose taste sensitivity using a conditioned taste aversion paradigm. Rats were given a high fat diet for 2 weeks and were assigned as obesity-prone (P, upper tertile) or obesity-resistant (R, lower tertile) based on weight gain. Each group was then given either chow (C, 10% fat) or the high fat diet (F, 46% fat) for the remainder of the experiment (similar to 18 weeks) such that there were four groups - obesity-prone on chow (C-P), obesity-prone on high fat (H-P), obesity-resistant on chow (C-R), obesity resistant on high fat (H-R). The sucrose sensitivity of phenotypic obesity-prone rats is lower than that of obesity-resistant rats in either H-fed or C-fed group, and all H-fed rats were more sensitivity than their C-fed counterparts (H-P vs. C-P; H-R vs. C-R). Body weight gain and total calories intake of phenotypic obesity-prone rats are more than that of obesity-resistant rats. The results suggest that lower sucrose taste sensitivity may contribute to body weight gain and total calories intake of phenotypic obesity prone rats compared to obesity-resistant rats, and there is correlation between the change in the sweet taste threshold and diet treatment. (C) 2017 Elsevier Inc. All rights reserved.

Keyword :

Sucrose taste sensitivity Conditioned taste aversion Taste detection thresholds Body weight

Cite:

Copy from the list or Export to your reference management。

GB/T 7714 Sun, Huiling , Yan, Junbao , Sun, Bo et al. Taste sensitivity to sucrose is lower in outbred Sprague-Dawley phenotypic obesity-prone rats than obesity-resistant rats [J]. | BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS , 2017 , 489 (2) : 155-163 .
MLA Sun, Huiling et al. "Taste sensitivity to sucrose is lower in outbred Sprague-Dawley phenotypic obesity-prone rats than obesity-resistant rats" . | BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS 489 . 2 (2017) : 155-163 .
APA Sun, Huiling , Yan, Junbao , Sun, Bo , Song, Lin , Yan, Jianqun . Taste sensitivity to sucrose is lower in outbred Sprague-Dawley phenotypic obesity-prone rats than obesity-resistant rats . | BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS , 2017 , 489 (2) , 155-163 .
Export to NoteExpress RIS BibTex
TrpC5 Mediates Acute Leptin and Serotonin Effects via Pomc Neurons SCIE PubMed
期刊论文 | 2017 , 18 (3) , 583-592 | CELL REPORTS | IF: 8.032
WoS CC Cited Count: 16
Abstract&Keyword Cite

Abstract :

The molecular mechanisms underlying acute leptin and serotonin 2C receptor-induced hypophagia remain unclear. Here, we show that neuronal and pro-opiomelanocortin (Pomc)-specific loss of transient receptor potential cation 5 (TrpC5) subunits is sufficient to decrease energy expenditure and increase food intake resulting in elevated body weight. Deficiency of Trpc5 subunits in Pomc neurons is also sufficient to block the anorexigenic effects of leptin and serotonin 2C receptor (Ht2Cr) agonists. The loss of acute anorexigenic effects of these receptors is concomitant with a blunted electrophysiological response to both leptin and Ht2Cr agonists in arcuate Pomc neurons. We also demonstrate that the Ht2Cr agonist lorcaserin-induced improvements in glucose and insulin tolerance are blocked by TrpC5 deficiency in Pomc neurons. Together, our results link TrpC5 subunits in the brain with leptinand serotonin 2C receptor-dependent changes in neuronal activity, as well as energy balance, feeding behavior, and glucose metabolism.

Cite:

Copy from the list or Export to your reference management。

GB/T 7714 Gao, Yong , Yao, Ting , Deng, Zhuo et al. TrpC5 Mediates Acute Leptin and Serotonin Effects via Pomc Neurons [J]. | CELL REPORTS , 2017 , 18 (3) : 583-592 .
MLA Gao, Yong et al. "TrpC5 Mediates Acute Leptin and Serotonin Effects via Pomc Neurons" . | CELL REPORTS 18 . 3 (2017) : 583-592 .
APA Gao, Yong , Yao, Ting , Deng, Zhuo , Sohn, Jong-Woo , Sun, Jia , Huang, Yiru et al. TrpC5 Mediates Acute Leptin and Serotonin Effects via Pomc Neurons . | CELL REPORTS , 2017 , 18 (3) , 583-592 .
Export to NoteExpress RIS BibTex
Cold Exposure Differentially Stimulates Angiogenesis in BAT and WAT of Mice: Implication in Adrenergic Activation SCIE PubMed Scopus
期刊论文 | 2017 , 42 (3) , 974-986 | CELLULAR PHYSIOLOGY AND BIOCHEMISTRY | IF: 5.5
WoS CC Cited Count: 2 SCOPUS Cited Count: 4
Abstract&Keyword Cite

Abstract :

Background/Aims: To characterize the temporal profile of cold-induced angiogenesis in brown and white adipose tissues of mice in vivo and the temporal changes of angiogenic factors in primary mice brown (BA) and white adipocytes (WA) treated with beta(3)-adrenoceptor agonist (CL316,243) in vitro. Methods: 8-week old male C57BL/6J mice were individually housed in conventional cages under cold exposure (4 degrees C) for 1, 2, 3, 4 and 5 days. Interscapular brown adipose tissue (iBAT), inguinal subcutaneous (sWAT) and epididymal white adipose tissues (eWAT) were harvested for immunohistochemical and gene expression analysis. In vitro, primary mice BA and WA treated with or without CL316,243 were harvested for gene expression and protein secretion analysis. Results: A combination of morphological and genetic (Vegfa, Vegfr2, Hif-1 alpha, Pai1 and Pedf) analyses demonstrated depot-specific angiogenesis in response to cold exposure. Upon CL316,243 treatment, angiogenic factors (Vegfa, Vegfr2, Hif-1 alpha, Pai1 and Pedf) and secreted protein VEGFA were transiently increased in both BA and WA. Conclusion: Our results show that iBAT is highly responsive to cold-induced angiogenesis that is mainly supported by sWAT with a lesser extent by eWAT. Moreover, the angiogenesis is a transient process with the angiogenic factors may work in an autocrine/paracrine manner. (C) 2017 The Author(s) Published by S. Karger AG, Basel

Keyword :

Cold exposure Time course Angiogenesis Adipose tissues Adrenergic activation

Cite:

Copy from the list or Export to your reference management。

GB/T 7714 Luo, Xiao , Jia, Ru , Luo, Xiao-qin et al. Cold Exposure Differentially Stimulates Angiogenesis in BAT and WAT of Mice: Implication in Adrenergic Activation [J]. | CELLULAR PHYSIOLOGY AND BIOCHEMISTRY , 2017 , 42 (3) : 974-986 .
MLA Luo, Xiao et al. "Cold Exposure Differentially Stimulates Angiogenesis in BAT and WAT of Mice: Implication in Adrenergic Activation" . | CELLULAR PHYSIOLOGY AND BIOCHEMISTRY 42 . 3 (2017) : 974-986 .
APA Luo, Xiao , Jia, Ru , Luo, Xiao-qin , Wang, Guan , Zhang, Qiang-ling , Qiao, Hu et al. Cold Exposure Differentially Stimulates Angiogenesis in BAT and WAT of Mice: Implication in Adrenergic Activation . | CELLULAR PHYSIOLOGY AND BIOCHEMISTRY , 2017 , 42 (3) , 974-986 .
Export to NoteExpress RIS BibTex
Ire1 alpha in Pomc Neurons Is Required for Thermogenesis and Glycemia SCIE PubMed
期刊论文 | 2017 , 66 (3) , 663-673 | DIABETES | IF: 7.273
WoS CC Cited Count: 6
Abstract&Keyword Cite

Abstract :

Whether neuronal inositol-requiring enzyme 1 (Ire1) is required for the proper regulation of energy balance and glucose homeostasis is unclear. We found that proopiomelanocortin (Pomc)-specific deficiency of Ire1 alpha accelerated diet-induced obesity concomitant with a decrease in energy expenditure. This hypometabolic phenotype included deficits in thermogenic responses to diet and cold exposure as well as "beiging" of white adipose tissue. We also demonstrate that loss of Ire1 alpha in Pomc neurons impaired whole-body glucose and insulin tolerance as well as hepatic insulin sensitivity. At the cellular level, deletion of Ire1 alpha in Pomc neurons elevated hypothalamic endoplasmic reticulum (ER) stress and predisposed Pomc neurons to leptin and insulin resistance. Together, the current studies extend and confirm conclusions that Ire1 alpha-Xbp1s and associated molecular targets link ER stress in arcuate Pomc neurons to aspects of normal energy and glucose homeostasis.

Cite:

Copy from the list or Export to your reference management。

GB/T 7714 Yao, Ting , Deng, Zhuo , Gao, Yong et al. Ire1 alpha in Pomc Neurons Is Required for Thermogenesis and Glycemia [J]. | DIABETES , 2017 , 66 (3) : 663-673 .
MLA Yao, Ting et al. "Ire1 alpha in Pomc Neurons Is Required for Thermogenesis and Glycemia" . | DIABETES 66 . 3 (2017) : 663-673 .
APA Yao, Ting , Deng, Zhuo , Gao, Yong , Sun, Jia , Kong, Xingxing , Huang, Yiru et al. Ire1 alpha in Pomc Neurons Is Required for Thermogenesis and Glycemia . | DIABETES , 2017 , 66 (3) , 663-673 .
Export to NoteExpress RIS BibTex
高脂饮食影响雌性大鼠对糖精溶液的喜好并改变多巴胺及阿片相关基因的表达 CSCD PKU
期刊论文 | 2016 , (6) | 西安交通大学学报(医学版)
Abstract&Keyword Cite

Keyword :

糖精溶液喜好率 高脂饮食 阿片肽及其受体 奖赏系统 多巴胺

Cite:

Copy from the list or Export to your reference management。

GB/T 7714 孙波 , 宋琳 , 罗肖 et al. 高脂饮食影响雌性大鼠对糖精溶液的喜好并改变多巴胺及阿片相关基因的表达 [J]. | 西安交通大学学报(医学版) , 2016 , (6) .
MLA 孙波 et al. "高脂饮食影响雌性大鼠对糖精溶液的喜好并改变多巴胺及阿片相关基因的表达" . | 西安交通大学学报(医学版) 6 (2016) .
APA 孙波 , 宋琳 , 罗肖 , 孟凯 , 闫剑群 . 高脂饮食影响雌性大鼠对糖精溶液的喜好并改变多巴胺及阿片相关基因的表达 . | 西安交通大学学报(医学版) , 2016 , (6) .
Export to NoteExpress RIS BibTex
Aldosterone induces rapid sodium intake by a nongenomic mechanism in the nucleus tractus solitarius SCIE PubMed Scopus
期刊论文 | 2016 , 6 | SCIENTIFIC REPORTS | IF: 4.259
WoS CC Cited Count: 1 SCOPUS Cited Count: 2
Abstract&Keyword Cite

Abstract :

The purpose of this study was to determine whether aldosterone has a rapid action in the nucleus tractus solitarius (NTS) that increases sodium intake, and to examine whether this effect of aldosterone, if present, is mediated by G protein-coupled estrogen receptor (GPER). Adult male Sprague-Dawley rats with a stainless-steel cannula in the NTS were used. Aldosterone was injected into the NTS at the doses of 1, 5, 10 and 20 ng 0.1 mu l(-1). A rapid dose-related increase of 0.3 M NaCl intake was induced within 30 min and this increase was not suppressed by the mineralocorticoid receptor (MR) antagonist spironolactone (10 ng 0.1 mu l(-1)). Water intake was not affected by aldosterone. The GPER agonist G-1 produced a parallel and significant increase in sodium intake, while pre-treatment with GPER antagonist G15 (10 ng 0.1 mu l(-1)) blocked the G-1 or aldosterone-induced rapid sodium intake. In addition, sodium intake induced by sodium depletion or low-sodium diet fell within 30 min after injection into the NTS of the MR antagonist spironolactone, while G15 had no effect. Our results confirm previous reports, and support the hypothesis that aldosterone evokes rapid sodium intake through a non-genomic mechanism involving GPER in NTS.

Cite:

Copy from the list or Export to your reference management。

GB/T 7714 Qiao, Hu , Hu, Bo , Zhou, Hong et al. Aldosterone induces rapid sodium intake by a nongenomic mechanism in the nucleus tractus solitarius [J]. | SCIENTIFIC REPORTS , 2016 , 6 .
MLA Qiao, Hu et al. "Aldosterone induces rapid sodium intake by a nongenomic mechanism in the nucleus tractus solitarius" . | SCIENTIFIC REPORTS 6 (2016) .
APA Qiao, Hu , Hu, Bo , Zhou, Hong , Yan, Jianqun , Jia, Ru , Lu, Bo et al. Aldosterone induces rapid sodium intake by a nongenomic mechanism in the nucleus tractus solitarius . | SCIENTIFIC REPORTS , 2016 , 6 .
Export to NoteExpress RIS BibTex
Adiponectin potentiates the acute effects of leptin in arcuate Pomc neurons SCIE PubMed
期刊论文 | 2016 , 5 (10) , 882-891 | MOLECULAR METABOLISM | IF: 6.799
WoS CC Cited Count: 15
Abstract&Keyword Cite

Abstract :

Objective: Adiponectin receptors (AdipoRs) are located on neurons of the hypothalamus involved in metabolic regulation - including arcuate proopiomelanocortin (Pomc) and Neuropeptide Y/Agouti-related peptide (NPY/AgRP) neurons. AdipoRs play a critical role in regulating glucose and fatty acid metabolism by initiating several signaling cascades overlapping with Leptin receptors (LepRs). However, the mechanism by which adiponectin regulates cellular activity in the brain remains undefined. Methods: In order to resolve this issue, we utilized neuron-specific transgenic mouse models to identify Pomc and NPY/AgRP neurons which express LepRs for patch-clamp electrophysiology experiments. Results: We found that leptin and adiponectin synergistically activated melanocortin neurons in the arcuate nucleus. Conversely, NPY/AgRP neurons were inhibited in response to adiponectin. The adiponectin-induced depolarization of arcuate Pomc neurons occurred via activation of Phosphoinositide-3-kinase (PI3K) signaling, independent of 5' AMP-activated protein kinase (AMPK) activity. Adiponectin also activated melanocortin neurons at various physiological glucose levels. Conclusions: Our results demonstrate a requirement for PI3K signaling in the acute adiponectin-induced effects on the cellular activity of arcuate melanocortin neurons. Moreover, these data provide evidence for PI3K as a substrate for both leptin and adiponectin to regulate energy balance and glucose metabolism via melanocortin activity. (C) 2016 The Author(s). Published by Elsevier GmbH.

Keyword :

Melanocortin Energy balance Electrophysiology Patch-clamp Obesity Diabetes

Cite:

Copy from the list or Export to your reference management。

GB/T 7714 Sun, Jia , Gao, Yong , Yao, Ting et al. Adiponectin potentiates the acute effects of leptin in arcuate Pomc neurons [J]. | MOLECULAR METABOLISM , 2016 , 5 (10) : 882-891 .
MLA Sun, Jia et al. "Adiponectin potentiates the acute effects of leptin in arcuate Pomc neurons" . | MOLECULAR METABOLISM 5 . 10 (2016) : 882-891 .
APA Sun, Jia , Gao, Yong , Yao, Ting , Huang, Yiru , He, Zhenyan , Kong, Xingxing et al. Adiponectin potentiates the acute effects of leptin in arcuate Pomc neurons . | MOLECULAR METABOLISM , 2016 , 5 (10) , 882-891 .
Export to NoteExpress RIS BibTex
Cold-Induced Browning Dynamically Alters the Expression Profiles of Inflammatory Adipokines with Tissue Specificity in Mice SCIE PubMed Scopus
期刊论文 | 2016 , 17 (5) | INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES | IF: 3.226
WoS CC Cited Count: 7 SCOPUS Cited Count: 8
Abstract&Keyword Cite

Abstract :

Cold exposure or beta(3)-adrenoceptor agonist treatment induces the adipose tissues remodeling, relevant for beige adipogenesis within white adipose tissue (WAT). It remains unclear whether this process influences inflammatory adipokines expression in adipose tissues. We determine the temporal profile of cold or beta(3)-adrenoceptor agonist (CL316,243)-induced changes in the expression of inflammatory adipokines in adipose tissues in mice or primary mice adipocytes. Male C57BL/6J mice at eight weeks old were exposed to 4 degrees C for 1-5 days. Interscapular brown adipose tissue (iBAT), inguinal subcutaneous WAT (sWAT) and epididymal WAT (eWAT) were harvested for gene and protein expression analysis. In addition, cultured primary mice brown adipocyte (BA) and white adipocyte (WA) treated with or without CL316,243 were harvested for gene expression analysis. The inflammatory adipokines expressed significantly higher in WAT than BAT at baseline. They were rapidly changed in iBAT, while down-regulated in sWAT and up-regulated in eWAT during the cold acclimation. Upon CL316,243 treatment, detected inflammatory adipokines except Leptin were transiently increased in both BA and WA. Our in vivo and in vitro data demonstrate that the browning process alters the inflammatory adipokines expression in adipose tissues, which is acutely responded to in iBAT, dynamically decreased in sWAT whilst increased in eWAT for compensation.

Keyword :

cold exposure beta(3)-adrenoceptor agonist depot-specificity beige cells inflammatory adipokines

Cite:

Copy from the list or Export to your reference management。

GB/T 7714 Luo, Xiao , Jia, Ru , Zhang, Qiangling et al. Cold-Induced Browning Dynamically Alters the Expression Profiles of Inflammatory Adipokines with Tissue Specificity in Mice [J]. | INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES , 2016 , 17 (5) .
MLA Luo, Xiao et al. "Cold-Induced Browning Dynamically Alters the Expression Profiles of Inflammatory Adipokines with Tissue Specificity in Mice" . | INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES 17 . 5 (2016) .
APA Luo, Xiao , Jia, Ru , Zhang, Qiangling , Sun, Bo , Yan, Jianqun . Cold-Induced Browning Dynamically Alters the Expression Profiles of Inflammatory Adipokines with Tissue Specificity in Mice . | INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES , 2016 , 17 (5) .
Export to NoteExpress RIS BibTex
10| 20| 50 per page
< Page ,Total 11 >

Export

Results:

Selected

to

Format:
FAQ| About| Online/Total:3624/50773320
Address:XI'AN JIAOTONG UNIVERSITY LIBRARY(No.28, Xianning West Road, Xi'an, Shaanxi Post Code:710049) Contact Us:029-82667865
Copyright:XI'AN JIAOTONG UNIVERSITY LIBRARY Technical Support:Beijing Aegean Software Co., Ltd.